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Detailed chapter

Hereditary Cancers — Peutz–Jeghers Syndrome

STK11, hamartomatous polyps, and cancer surveillance

Peutz–Jeghers syndrome (PJS) is linked to STK11 germline changes. Hamartomatous polyps (often small bowel), characteristic mucocutaneous pigmentation, and raised GI/extra-GI cancer risk frame the syndrome. Surgery is often driven by obstruction, intussusception, or bleeding rather than “clear every polyp in one operation.”

Peutz–Jeghers · educational illustration

Educational schematic. Some structures may be simplified or emphasized for clarity; not an exact anatomical depiction.

© Cengiz Dibekoğlu — illustrative; not for unauthorized use

Figure summary

This educational figure shows: Peutz–Jeghers.

Key points

  • Clinical clues

    Recurrent small-bowel polyps/intussusception, typical pigmentation, family PJS history, or a known STK11 mutation open evaluation.

  • Surgical role

    Key note inside

    Goals focus on treating complications while preserving intestine. A single “finish all polyps” operation is rarely realistic.

  • Surveillance and family

    Organ-specific surveillance intervals follow guidelines and counseling. Cascade testing is offered to relatives.

  • Important

    Key note inside

    This page is for education. It is not medical advice and does not replace a visit with your physician.

Frequently asked questions

  • Are freckles around the mouth always PJS?

    No. Diagnosis needs clinical context plus counseling/genetic confirmation when indicated.

  • Can one surgery remove all polyps?

    Usually not. Endoscopy and surgery are sequenced to treat complications while preserving bowel length.

This page is for education. It is not medical advice and does not replace a visit with your physician.

Medical editor: Assoc. Prof. Cengiz Dibekoğlu, MD · Last medically reviewed: August 2026 · English patient-education chapter.